Distribution, connection and function of ALDH1A1+/TH+ neurons in substantia nigra pars reticulata of mouse
Open Access
- 14 November 2023
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 818, 137555
Abstract
No abstract availableKeywords
Funding Information
- Natural Science Foundation of Guangdong Province (2023A1515012780)
- Guangzhou Municipal Science and Technology Program key projects (202007030001)
- Guangdong Provincial Department of Science and Technology (2020A1414040027)
- National Natural Science Foundation of China (82,071,425)
This publication has 39 references indexed in Scilit:
- Determinants of buildup of the toxic dopamine metaboliteDOPALin Parkinson's diseaseJournal of Neurochemistry, 2013
- Pathological α-Synuclein Transmission Initiates Parkinson-like Neurodegeneration in Nontransgenic MiceScience, 2012
- Conditional Expression of Parkinson's Disease-Related Mutant -Synuclein in the Midbrain Dopaminergic Neurons Causes Progressive Neurodegeneration and Degradation of Transcription Factor Nuclear Receptor Related 1Journal of Neuroscience, 2012
- Aldehyde dehydrogenases are regulators of hematopoietic stem cell numbers and B-cell developmentExperimental Hematology, 2012
- Otx2 controls neuron subtype identity in ventral tegmental area and antagonizes vulnerability to MPTPNature Neuroscience, 2010
- Extensive enteric nervous system abnormalities in mice transgenic for artificial chromosomes containing Parkinson disease-associated α-synuclein gene mutations precede central nervous system changesHuman Molecular Genetics, 2010
- Parkinson Phenotype in Aged PINK1-Deficient Mice Is Accompanied by Progressive Mitochondrial Dysfunction in Absence of NeurodegenerationPLOS ONE, 2009
- Parkinson's disease mutations in PINK1 result in decreased Complex I activity and deficient synaptic functionEMBO Molecular Medicine, 2009
- Small Intestinal Bacterial Overgrowth: Roles of Antibiotics, Prebiotics, and ProbioticsGastroenterology, 2006
- Idiopathic Parkinson's disease: possible routes by which vulnerable neuronal types may be subject to neuroinvasion by an unknown pathogenJournal of Neural Transmission, 2003