PI3K signaling pathway in normal B cells and indolent B-cell malignancies
Authors:
- 1 December 2016
- journal article
- review article
- Published by Elsevier in Seminars in Oncology
- Vol. 43 (6) , 647-654
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- RAS and RHO Families of GTPases Directly Regulate Distinct Phosphoinositide 3-Kinase IsoformsCell, 2013
- Signaling by the Phosphoinositide 3-Kinase Family in Immune CellsAnnual Review of Immunology, 2013
- Receptor Tyrosine Kinases and TLR/IL1Rs Unexpectedly Activate Myeloid Cell PI3Kγ, A Single Convergent Point Promoting Tumor Inflammation and ProgressionCancer Cell, 2011
- Hallmarks of Cancer: The Next GenerationCell, 2011
- Phosphatidylinositol 3-kinase-δ inhibitor CAL-101 shows promising preclinical activity in chronic lymphocytic leukemia by antagonizing intrinsic and extrinsic cellular survival signalsBlood, 2010
- The PI3K Isoforms p110α and p110δ Are Essential for Pre–B Cell Receptor Signaling and B Cell DevelopmentScience Signaling, 2010
- Introduction2010
- BAFF controls B cell metabolic fitness through a PKCβ- and Akt-dependent mechanismJournal of Experimental Medicine, 2006
- ZAP-70 expression identifies a chronic lymphocytic leukemia subtype with unmutated immunoglobulin genes, inferior clinical outcome, and distinct gene expression profileBlood, 2003
- Central Role for G Protein-Coupled Phosphoinositide 3-Kinase γ in InflammationScience, 2000