Gα12 overexpression induced by miR-16 dysregulation contributes to liver fibrosis by promoting autophagy in hepatic stellate cells
Authors:
- 1 March 2018
- journal article
- research article
- Published by Elsevier in Journal of Hepatology
- Vol. 68 (3) , 493-504
Abstract
No abstract availableKeywords
Funding Information
- NRF
- MSIP (NRF-2015R1A2A1A10052663)
- NRF-JSPS program
This publication has 42 references indexed in Scilit:
- Autophagy Releases Lipid That Promotes Fibrogenesis by Activated Hepatic Stellate Cells in Mice and in Human TissuesGastroenterology, 2012
- Autophagy in mammalian development and differentiationNature Cell Biology, 2010
- ANG II promotes autophagy in podocytesAmerican Journal of Physiology-Cell Physiology, 2010
- Downregulation of hepatic stellate cell activation by retinol and palmitate mediated by adipose differentiation‐related protein (ADRP)Journal of Cellular Physiology, 2010
- Liver fibrosis causes downregulation of miRNA-150 and miRNA-194 in hepatic stellate cells, and their overexpression causes decreased stellate cell activationAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2010
- Modulation of Hepatic Fibrosis by c-Jun-N-Terminal Kinase InhibitionGastroenterology, 2010
- Hepatic Stellate Cells: Protean, Multifunctional, and Enigmatic Cells of the LiverPhysiological Reviews, 2008
- The Atg12-Atg5 Conjugate Has a Novel E3-like Activity for Protein Lipidation in AutophagyJournal of Biological Chemistry, 2007
- MicroRNA Targeting Specificity in Mammals: Determinants beyond Seed PairingMolecular Cell, 2007
- LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processingThe EMBO Journal, 2000