Cosmc loss induces truncated O-glycosylation and accelerates Kras-driven pancreatic carcinogenesis
Authors:
- Baris MercanogluBaris Mercanoglu
- Nina SchrapsNSNina Schraps
- Anastasios D. GiannouAGAnastasios D. Giannou
- Jingxuan ZhouJZJingxuan Zhou
- Simon KindSKSimon Kind
- Maya ReineckeMRMaya Reinecke
- Karl-Frederick KarstensKarl-Frederick Karstens
- Benjamin DreyerBenjamin Dreyer
- Sönke HarderSHSönke Harder
- Siwen ZhangSZSiwen Zhang
- Eleftherios D. PapazoglouEPEleftherios D. Papazoglou
- Cenap GüngörCGCenap Güngör
Open Access
Abstract
No abstract availableFunding Information
- Deutsche Forschungsgemeinschaft (275533756)
This publication has 44 references indexed in Scilit:
- High core 1β1,3-galactosyltransferase 1 expression is associated with poor prognosis and promotes cellular radioresistance in lung adenocarcinomaJournal of Cancer Research and Clinical Oncology, 2024
- Characterization of the genomic landscape in large-scale Chinese patients with pancreatic cancerEBioMedicine, 2022
- Modeling pancreatic cancer in mice for experimental therapeuticsBiochimica et Biophysica Acta (BBA) - Reviews on Cancer, 2021
- Gene Set Knowledge Discovery with EnrichrCurrent Protocols, 2021
- Differences between KC and KPC pancreatic ductal adenocarcinoma mice models, in terms of their modeling biology and their clinical relevancePancreatology, 2020
- Pancreatic Ductal Adenocarcinoma and Its Precursor LesionsThe American Journal of Pathology, 2019
- Lectin Histochemistry for Metastasizing and Non-metastasizing Cancer Cells2017
- Tissue Microarrays2016
- Origin of pancreatic ductal adenocarcinoma from atypical flat lesions: a comparative study in transgenic mice and human tissuesThe Journal of Pathology, 2011
- Trp53R172H and KrasG12D cooperate to promote chromosomal instability and widely metastatic pancreatic ductal adenocarcinoma in miceCancer Cell, 2005