Exosomes derived from cardiac fibroblasts with angiotensin II stimulation provoke hypertrophy and autophagy inhibition in cardiomyocytes
- 10 October 2023
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 682, 199-206
Abstract
No abstract availableKeywords
Funding Information
- Basic and Applied Basic Research Foundation of Guangdong Province (2020A1515011512)
- National Natural Science Foundation of China (82070268)
- Sun Yat-sen University (18ykpy26)
This publication has 39 references indexed in Scilit:
- A critical role of cardiac fibroblast-derived exosomes in activating renin angiotensin system in cardiomyocytesJournal of Molecular and Cellular Cardiology, 2015
- p62/SQSTM1 functions as a signaling hub and an autophagy adaptorThe FEBS Journal, 2015
- Cardiac fibroblast–derived microRNA passenger strand-enriched exosomes mediate cardiomyocyte hypertrophyJournal of Clinical Investigation, 2014
- Cardiac fibroblasts protect cardiomyocytes against lethal ischemia–reperfusion injuryJournal of Molecular and Cellular Cardiology, 2014
- Angiogenesis and Cardiac HypertrophyCirculation Research, 2014
- Enhanced autophagy ameliorates cardiac proteinopathyJournal of Clinical Investigation, 2013
- Atg7 Induces Basal Autophagy and Rescues Autophagic Deficiency in CryAB R120G CardiomyocytesCirculation Research, 2011
- Inhibition of autophagy in the heart induces age-related cardiomyopathyAutophagy, 2010
- Cardiac fibroblasts: At the heart of myocardial remodelingPharmacology & Therapeutics, 2009
- The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stressNature Medicine, 2007