Cognitive dysfunction in diabetic rats is prevented by pyridoxamine treatment. A multidisciplinary investigation
Open Access
- 1 October 2019
- journal article
- research article
- Published by Elsevier in Molecular Metabolism
- Vol. 28, 107-119
Abstract
No abstract availableKeywords
Funding Information
- Medical Research Council
This publication has 51 references indexed in Scilit:
- The novel object recognition memory: neurobiology, test procedure, and its modificationsCognitive Processing, 2011
- Simultaneous analysis of relative protein expression levels across multiple samples using iTRAQ isobaric tags with 2D nano LC–MS/MSNature Protocols, 2010
- Development and Performance of a Gas Chromatography−Time-of-Flight Mass Spectrometry Analysis for Large-Scale Nontargeted Metabolomic Studies of Human SerumAnalytical Chemistry, 2009
- Defective insulin signaling pathway and increased glycogen synthase kinase‐3 activity in the brain of diabetic mice: Parallels with Alzheimer's disease and correction by insulinJournal of Neuroscience Research, 2008
- Synaptopodin maintains the neural activity-dependent enlargement of dendritic spines in hippocampal neuronsMolecular and Cellular Neuroscience, 2008
- Cognitive Dysfunction and Diabetes MellitusEndocrine Reviews, 2008
- Synaptopodin, a molecule involved in the formation of the dendritic spine apparatus, is a dual actin/α‐actinin binding proteinJournal of Neurochemistry, 2004
- Neuronal nicotinic receptors: from structure to pathologyProgress in Neurobiology, 2004
- Pyridoxamine inhibits early renal disease and dyslipidemia in the streptozotocin-diabetic ratKidney International, 2002
- β-Amyloid produces a delayed NMDA receptor- dependent reduction in synaptic transmission in rat hippocampusNeuroReport, 1996