Methylmercury activates ASK1/JNK signaling pathways, leading to apoptosis due to both mitochondria- and endoplasmic reticulum (ER)-generated processes in myogenic cell lines

This publication has 41 references indexed in Scilit:

• Caspase-12 processing and fragment translocation into nuclei of tunicamycin-treated cells
  Cell Death & Differentiation, 2002
• Time course assessment of methylmercury effects on C6 glioma cells: submicromolar concentrations induce oxidative DNA damage and apoptosis
  Journal of Neuroscience Research, 2002
• Overexpression of the Ubiquitin-Conjugating Enzyme Cdc34 Confers Resistance to Methylmercury in Saccharomyces cerevisiae
  Molecular Pharmacology, 2002
• Ubiquitin‐proteasome system is responsible for the protection of yeast and human cells against methylmercury
  The FASEB Journal, 2002
• Apoptotic morphology does not always require caspase activity in rat cerebellar granule neurons
  Neurotoxicity Research, 2001
• Methylmercury and H2O2 provoke lysosomal damage in human astrocytoma D384 cells followed by apoptosis
  Free Radical Biology and Medicine, 2001
• Activation of Mitogen-activated Protein Kinase by H2O2
  Journal of Biological Chemistry, 1996
• [21] Thioredoxin and thioredoxin reductase
  1995
• Differential regulation of rat liver selenoprotein mRNAs in selenium deficiency
  Biochemical and Biophysical Research Communications, 1992
• Increase in metallothionein produced by chemicals that induce oxidative stress
  Toxicology and Applied Pharmacology, 1991