SMEAF attenuates the production of pro-inflammatory mediators through the inactivation of Akt-dependent NF-κB, p38 and ERK1/2 pathways in LPS-stimulated BV-2 microglial cells

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Abstract
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Funding Information
  • University of Malaya (UM.C/625/1/HIR/175)
  • Ministry of Science, Technology and Innovation, Malaysia (MO001-2014, PG11820-2013A, 02-02-10-SF0215)

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