Both LRP5 and LRP6 Receptors Are Required to Respond to Physiological Wnt Ligands in Mammary Epithelial Cells and Fibroblasts

This publication has 66 references indexed in Scilit:

• Wnt Signaling in Mammary Glands: Plastic Cell Fates and Combinatorial Signaling
  Cold Spring Harbor Perspectives in Biology, 2012
• β-Catenin pathway activation in breast cancer is associated with triple-negative phenotype but not with CTNNB1 mutation
  Laboratory Investigation, 2011
• Wnt Isoform-Specific Interactions with Coreceptor Specify Inhibition or Potentiation of Signaling by LRP6 Antibodies
  PLOS ONE, 2010
• Towards an integrated view of Wnt signaling in development
  Development, 2009
• WNT signaling enhances breast cancer cell motility and blockade of the WNT pathway by sFRP1 suppresses MDA-MB-231 xenograft growth
  Breast Cancer Research, 2009
• Frequent epigenetic inactivation of Wnt antagonist genes in breast cancer
  British Journal of Cancer, 2008
• Analysis of Endogenous LRP6 Function Reveals a Novel Feedback Mechanism by Which Wnt Negatively Regulates Its Receptor
  Molecular and Cellular Biology, 2007
• R-spondin1 Is a High Affinity Ligand for LRP6 and Induces LRP6 Phosphorylation and β-Catenin Signaling
  Journal of Biological Chemistry, 2007
• Role of the intracellular domains of LRP5 and LRP6 in activating the Wnt canonical pathway
  Journal of Cellular Biochemistry, 2005
• Armadillo/β-catenin signals in the nucleus – proof beyond a reasonable doubt?
  Nature Cell Biology, 2003