Consensus draft of the native mouse podocyte-ome
- 1 August 2022
- journal article
- research article
- Published by American Physiological Society in American Journal of Physiology-Renal Physiology
- Vol. 323 (2) , F182-F197
Abstract
Podocytes are key components of glomerular filtration and are affected in various kidney diseases. Here, we present an integrated, robust definition of molecular identity across proteomic, single-cell transcriptomics, and bulk transcriptomic studies on native mouse podocytes. We created the “PodIdent” app, a novel knowledge base promoting access to the presence and expression of specific proteins for podocytes.Keywords
This publication has 72 references indexed in Scilit:
- Pathview: an R/Bioconductor package for pathway-based data integration and visualizationBioinformatics, 2013
- 1D and 2D annotation enrichment: a statistical method integrating quantitative proteomics with complementary high-throughput dataBMC Bioinformatics, 2012
- Podocyte-Specific Deletion of Myh9 Encoding Nonmuscle Myosin Heavy Chain 2A Predisposes Mice to GlomerulopathyMolecular and Cellular Biology, 2011
- Urine Podocyte mRNAs Mark Progression of Renal DiseaseJournal of the American Society of Nephrology, 2009
- RNA-seq: An assessment of technical reproducibility and comparison with gene expression arraysGenome Research, 2008
- TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal functionNature Genetics, 2005
- The role of podocytes in glomerular pathobiologyClinical and Experimental Nephrology, 2003
- Mass spectrometry-based proteomicsNature, 2003
- KEGG: Kyoto Encyclopedia of Genes and GenomesNucleic Acids Research, 2000
- Positionally Cloned Gene for a Novel Glomerular Protein—Nephrin—Is Mutated in Congenital Nephrotic SyndromeMolecular Cell, 1998