NRF2/KEAP1 and Wnt/β‐catenin in the multistep process of liver carcinogenesis in humans and rats
Open Access
- 4 April 2015
- journal article
- editorial
- Published by Wolters Kluwer Health in Hepatology
- Vol. 62 (3) , 677-679
Abstract
No abstract availableKeywords
Funding Information
- Inca (ICGC)
- Ligue Contre le Cancer (équipe Labellisée)
This publication has 10 references indexed in Scilit:
- Exome sequencing of hepatocellular carcinomas identifies new mutational signatures and potential therapeutic targetsNature Genetics, 2015
- Nrf2, but not β‐catenin, mutation represents an early event in rat hepatocarcinogenesisHepatology, 2015
- The genomic landscape of hepatoblastoma and their progenies with HCC-like featuresJournal of Hepatology, 2014
- Trans-ancestry mutational landscape of hepatocellular carcinoma genomesNature Genetics, 2014
- Telomerase reverse transcriptase promoter mutation is an early somatic genetic alteration in the transformation of premalignant nodules in hepatocellular carcinoma on cirrhosisHepatology, 2014
- High frequency of telomerase reverse-transcriptase promoter somatic mutations in hepatocellular carcinoma and preneoplastic lesionsNature Communications, 2013
- NRF2 and cancer: the good, the bad and the importance of contextNature Reviews Cancer, 2012
- Integrated analysis of somatic mutations and focal copy-number changes identifies key genes and pathways in hepatocellular carcinomaNature Genetics, 2012
- Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesisNature, 2011
- Sensitivity to carcinogenesis is increased and chemoprotective efficacy of enzyme inducers is lost in nrf2 transcription factor-deficient miceProceedings of the National Academy of Sciences, 2001