Mechanisms underlying mutually exclusive expression of virulence genes by malaria parasites

This publication has 23 references indexed in Scilit:

• A Silenced Plasmodium falciparum var Promoter Can Be Activated In Vivo through Spontaneous Deletion of a Silencing Element in the Intron
  Eukaryotic Cell, 2005
• One neuron–one receptor rule in the mouse olfactory system
  Trends in Genetics, 2004
• A pol I transcriptional body associated with VSG mono-allelic expression in Trypanosoma brucei
  Nature, 2001
• Antigenic Variation at the Infected Red Cell Surface in Malaria
  Annual Review of Microbiology, 2001
• A functional role for Tsix transcription in blocking Xist RNA accumulation but not in X-chromosome choice
  Proceedings of the National Academy of Sciences, 2001
• Transformation of malaria parasites by the spontaneous uptake and expression of DNA from human erythrocytes
  Nucleic Acids Research, 2001
• Epigenetic reprogramming of the genome--from the germ line to the embryo and back again
  The International Journal of Developmental Biology, 2001
• Frequent ectopic recombination of virulence factor genes in telomeric chromosome clusters of P. falciparum
  Nature, 2000
• Subnuclear localization of the active variant surface glycoprotein gene expression site in Trypanosoma brucei
  Proceedings of the National Academy of Sciences, 1998
• Antigenic variation in malaria: in situ switching, relaxed and mutually exclusive transcription of var genes during intra‐erythrocytic development in Plasmodium falciparum
  The EMBO Journal, 1998