Restoration of SERCA ATPase prevents oxidative stress-related muscle atrophy and weakness
Open Access
- 1 January 2019
- journal article
- research article
- Published by Elsevier in Redox Biology
- Vol. 20, 68-74
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (AG051442-02O)
- U.S. Department of Veterans Affairs
This publication has 39 references indexed in Scilit:
- Muscle RANK is a key regulator of Ca2+ storage, SERCA activity, and function of fast-twitch skeletal musclesAmerican Journal of Physiology-Cell Physiology, 2016
- Introduction to Oxidative Stress in Biomedical and Biological ResearchBiomolecules, 2015
- Dysregulation of calcium homeostasis in muscular dystrophiesExpert Reviews in Molecular Medicine, 2014
- Dysregulation of Mitochondrial Quality Control Processes Contribute to Sarcopenia in a Mouse Model of Premature AgingPLOS ONE, 2013
- Hsp72 preserves muscle function and slows progression of severe muscular dystrophyNature, 2012
- Increased superoxide in vivo accelerates age‐associated muscle atrophy through mitochondrial dysfunction and neuromuscular junction degenerationThe FASEB Journal, 2009
- Reduction of mitochondrial H2O2 by overexpressing peroxiredoxin 3 improves glucose tolerance in miceAging Cell, 2008
- Denervation-induced skeletal muscle atrophy is associated with increased mitochondrial ROS productionAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2007
- Ca2+ Signaling in HEK-293 and Skeletal Muscle Cells Expressing Recombinant Ryanodine Receptors Harboring Malignant Hyperthermia and Central Core Disease MutationsJournal of Biological Chemistry, 2005
- Reactive oxygen species reduce myofibrillar Ca2+ sensitivity in fatiguing mouse skeletal muscle at 37°CThe Journal of Physiology, 2005