Prolyl hydroxylase inhibition protects the kidneys from ischemia via upregulation of glycogen storage
- 1 April 2020
- journal article
- research article
- Published by Elsevier in Kidney International
- Vol. 97 (4) , 687-701
Abstract
No abstract availableKeywords
Funding Information
- Japan Society for the Promotion of Science
This publication has 47 references indexed in Scilit:
- Multiplex Genome Engineering Using CRISPR/Cas SystemsScience, 2013
- Preischemic targeting of HIF prolyl hydroxylation inhibits fibrosis associated with acute kidney injuryAmerican Journal of Physiology-Renal Physiology, 2012
- Hypoxia Promotes Glycogen Accumulation through Hypoxia Inducible Factor (HIF)-Mediated Induction of Glycogen Synthase 1PLOS ONE, 2010
- Inhibition of lactate dehydrogenase A induces oxidative stress and inhibits tumor progressionProceedings of the National Academy of Sciences, 2010
- Hypoxia-Induced Autophagy Is Mediated through Hypoxia-Inducible Factor Induction of BNIP3 and BNIP3L via Their BH3 DomainsMolecular and Cellular Biology, 2009
- Effects of glutathione reductase inhibition on cellular thiol redox state and related systemsArchives of Biochemistry and Biophysics, 2009
- PARP-1 Inhibits Glycolysis in Ischemic KidneysJournal of the American Society of Nephrology, 2009
- Mitochondrial Autophagy Is an HIF-1-dependent Adaptive Metabolic Response to HypoxiaJournal of Biological Chemistry, 2008
- Inhibition of Hypoxia Inducible Factor Hydroxylases Protects Against Renal Ischemia-Reperfusion InjuryJournal of the American Society of Nephrology, 2008
- Acute Kidney Injury, Mortality, Length of Stay, and Costs in Hospitalized PatientsJournal of the American Society of Nephrology, 2005