Type I IFN signaling in the absence of IRGM1 promotes M. tuberculosis replication in immune cells by suppressing T cell responses

This publication has 89 references indexed in Scilit:

• Early cellular mechanisms of type I interferon-driven susceptibility to tuberculosis
  Cell, 2023
• Overview of STING-Associated Vasculopathy with Onset in Infancy (SAVI) Among 21 Patients
  The Journal of Allergy and Clinical Immunology: In Practice, 2021
• STING Gain-of-Function Disrupts Lymph Node Organogenesis and Innate Lymphoid Cell Development in Mice
  Cell Reports, 2020
• STING-associated lung disease in mice relies on T cells but not type I interferon
  Journal of Allergy and Clinical Immunology, 2019
• Alveolar Macrophages Provide an Early Mycobacterium tuberculosis Niche and Initiate Dissemination
  Cell Host & Microbe, 2018
• The impact of ISGylation during Mycobacterium tuberculosis infection in mice
  Microbes and Infection, 2017
• Type I Interferons Link Viral Infection to Enhanced Epithelial Turnover and Repair
  Cell Host & Microbe, 2015
• Defining features of protective CD4 T cell responses to Mycobacterium tuberculosis
  Current Opinion in Immunology, 2014
• Cell-to-Cell Transfer of M. tuberculosis Antigens Optimizes CD4 T Cell Priming
  Cell Host & Microbe, 2014
• Type I IFN signaling triggers immunopathology in tuberculosis‐susceptible mice by modulating lung phagocyte dynamics
  European Journal of Immunology, 2014