KLF4 prevents epithelial to mesenchymal transition in human corneal epithelial cells via endogenous TGF-β2 suppression
Authors:
Open Access
- 1 December 2019
- journal article
- research article
- Published by Elsevier in Regenerative Therapy
- Vol. 11, 249-257
Abstract
No abstract availableKeywords
Funding Information
- Osaka University
This publication has 34 references indexed in Scilit:
- Krüppel-Like Factor 4 Acts as an Oncogene in Colon Cancer Stem Cell-Enriched Spheroid CellsPLOS ONE, 2013
- Activation of Smad‐mediated TGF‐β signaling triggers epithelial–mesenchymal transitions in murine cloned corneal progenitor cellsJournal of Cellular Physiology, 2012
- Regulation of Corneal Epithelial Barrier Function by Krüppel-like Transcription Factor 4Investigative Ophthalmology & Visual Science, 2011
- Kruppel-like factor 4 (KLF4) is required for maintenance of breast cancer stem cells and for cell migration and invasionOncogene, 2011
- TGFβ in CancerCell, 2008
- Conditional Deletion of the Mouse Klf4 Gene Results in Corneal Epithelial Fragility, Stromal Edema, and Loss of Conjunctival Goblet CellsMolecular and Cellular Biology, 2007
- Induction of Pluripotent Stem Cells from Mouse Embryonic and Adult Fibroblast Cultures by Defined FactorsCell, 2006
- The keratocyte: Corneal stromal cell with variable repair phenotypesThe International Journal of Biochemistry & Cell Biology, 2006
- Transcriptional Profiling of Krüppel-like Factor 4 Reveals a Function in Cell Cycle Regulation and Epithelial DifferentiationJournal of Molecular Biology, 2003
- TGF-beta1 and Ha-Ras collaborate in modulating the phenotypic plasticity and invasiveness of epithelial tumor cells.Genes & Development, 1996