Long-term Exposure to Nicotine, via Ras Pathway, Induces Cyclin D1 to Stimulate G1 Cell Cycle Transition

This publication has 46 references indexed in Scilit:

• p53 Is Necessary for the Apoptotic Response Mediated by a Transient Increase of Ras Activity
  Molecular and Cellular Biology, 2002
• Regulation of Ras Signaling Specificity by Protein Kinase C
  Molecular and Cellular Biology, 2001
• ras activity and cyclin D1 expression: An essential mechanism of mouse skin tumor development
  Molecular Carcinogenesis, 1999
• Signalling pathways involved in nicotine regulation of apoptosis of human lung cancer cells
  Carcinogenesis, 1998
• Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a
  Cell, 1997
• Effects of nicotine on the nucleus accumbens and similarity to those of addictive drugs
  Nature, 1996
• Down-regulation of Platelet-derived Growth Factor Receptor Expression during Terminal Differentiation of 3T3-L1 Pre-adipocyte Fibroblasts
  Journal of Biological Chemistry, 1996
• Proteins regulating Ras and its relatives
  Nature, 1993
• Pharmacologic Aspects of Cigarette Smoking and Nicotine Addiction
  New England Journal of Medicine, 1988
• ras GENES
  Annual Review of Biochemistry, 1987