Retinal ON- and OFF-pathway dysfunction due to HCN channel inhibition evaluated using dark and light adapted flicker electroretinograms in rats
Authors:
- 12 June 2026
- journal article
- Published by Elsevier in Experimental Eye Research
- Vol. 270, 111126
Abstract
No abstract availableKeywords
Funding Information
- Dainippon Sumitomo Pharma
This publication has 13 references indexed in Scilit:
- Exploring HCN channels as novel drug targetsNature Reviews Drug Discovery, 2011
- HCN1 Channels Significantly Shape Retinal Photoresponses2011
- Modulation of rod photoreceptor output by HCN1 channels is essential for regular mesopic cone visionNature Communications, 2011
- Effect of HCN Channel Inhibition on Retinal Morphology and Function in Normal and Dystrophic RodentsInvestigative Ophthalmology & Visual Science, 2010
- Light responses in the mouse retina are prolonged upon targeted deletion of the HCN1 channel geneEuropean Journal of Neuroscience, 2008
- Postreceptoral contributions to the light-adapted ERG of mice lacking b-wavesExperimental Eye Research, 2008
- Differential distribution of hyperpolarization‐activated and cyclic nucleotide‐gated channels in cone bipolar cells of the rat retinaJournal of Comparative Neurology, 2007
- HCN channels are expressed differentially in retinal bipolar cells and concentrated at synaptic terminalsEuropean Journal of Neuroscience, 2003
- Temporal Properties of the Mouse Cone ElectroretinogramJournal of Neurophysiology, 2002
- Cellular expression and functional characterization of four hyperpolarization‐activated pacemaker channels in cardiac and neuronal tissuesEuropean Journal of Biochemistry, 2001