Malvidin protects against lipopolysaccharide-induced acute liver injury in mice via regulating Nrf2 and NLRP3 pathways and suppressing apoptosis and autophagy
- 2 September 2022
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 933, 175252
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (81773968)
- Priority Academic Program Development of Jiangsu Higher Education Institutions
This publication has 35 references indexed in Scilit:
- Expert panel assessment of acute liver injury identification in observational dataResearch in Social and Administrative Pharmacy, 2014
- NLRP3 inflammasome activation in d-galactosamine and lipopolysaccharide-induced acute liver failure: Role of heme oxygenase-1Free Radical Biology and Medicine, 2013
- Regulation of oxidative stress by Nrf2 in the pathophysiology of infectious diseasesMedecine Et Maladies Infectieuses, 2013
- The central role of initiator caspase-9 in apoptosis signal transduction and the regulation of its activation and activity on the apoptosomeExperimental Cell Research, 2012
- Regulation of autophagy by ROS: physiology and pathologyTrends in Biochemical Sciences, 2011
- Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilizationNature Immunology, 2008
- Autophagy in the Pathogenesis of DiseaseCell, 2008
- ROS, mitochondria and the regulation of autophagyTrends in Cell Biology, 2007
- Etoposide Prevents Apoptosis in Mouse Liver With D–Galactosamine/Lipopolysaccharide–Induced Fulminant Hepatic Failure Resulting in Reduction of LethalityHepatology, 2001
- U-Shaped Dose-Responses in Biology, Toxicology, and Public HealthAnnual Review of Public Health, 2001