Macrophages signal cross-talks under hyperglycemic conditions and their link to cyclophilins

This publication has 67 references indexed in Scilit:

• Cyclophilin A and C are the Main Components of Extracellular Vesicles in Response to Hyperglycemia in BV2 Microglial Cells
  Molecular Neurobiology, 2025
• SIRT1 ameliorated septic associated-lung injury and macrophages apoptosis via inhibiting endoplasmic reticulum stress
  Cellular Signalling, 2022
• Metformin alleviates high glucose-induced ER stress and inflammation by inhibiting the interaction between caveolin1 and AMPKα in rat astrocytes
  Biochemical and Biophysical Research Communications, 2021
• ERK1/2 activated PHLPP1 induces skeletal muscle ER stress through the inhibition of a novel substrate AMPK
  Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2018
• AMPK signalling in health and disease
  Current Opinion in Cell Biology, 2017
• Extracellular cyclophilins in health and disease
  Biochimica et Biophysica Acta (BBA) - General Subjects, 2015
• High glucose activates Raw264.7 macrophages through RhoA kinase-mediated signaling pathway
  Cellular Signalling, 2015
• Proteomic profiling of high glucose primed monocytes identifies cyclophilin A as a potential secretory marker of inflammation in type 2 diabetes
  Proteomics, 2012
• Release of overexpressed CypB activates ERK signaling through CD147 binding for hepatoma cell resistance to oxidative stress
  Apoptosis, 2012
• Ginsenoside Rg2 induces orphan nuclear receptor SHP gene expression and inactivates GSK3β via AMP-activated protein kinase to inhibit hepatic glucose production in HepG2 cells
  Chemico-Biological Interactions, 2012