Carnosol, a Coffea arabica phenolic diterpene, has attracted attention as a multi-target anticancer compound. In cervical cancer models, it appears to regulate cancer-related gene expression, suppress proliferation, promote apoptosis, and reduce invasion-related behavior. Supporting evidence from other tumor systems shows effects on major oncogenic pathways, including NF-κB, MAPK/ERK, p38, PI3K/AKT/mTOR, p53/MDM2, and Nrf2. However, the current evidence remains preclinical, and clinical benefit in metastatic cervical cancer has not been established.