DPP-4 inhibition has beneficial effects on the heart after myocardial infarction
Authors:
- 1 February 2016
- journal article
- research article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 91, 72-80
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Dipeptidyl peptidase-4 inhibitors and GLP-1 reduce myocardial infarct size in a glucose-dependent mannerCardiovascular Diabetology, 2013
- DPP4 Deficiency Exerts Protective Effect against H2O2 Induced Oxidative Stress in Isolated CardiomyocytesPLOS ONE, 2013
- Intestinal CX 3 C chemokine receptor 1 high (CX 3 CR1 high ) myeloid cells prevent T-cell-dependent colitisProceedings of the National Academy of Sciences, 2012
- SDF-1/CXCR4 mediates acute protection of cardiac function through myocardial STAT3 signaling following global ischemia/reperfusion injuryAmerican Journal of Physiology-Heart and Circulatory Physiology, 2011
- Early and late effects of the DPP-4 inhibitor vildagliptin in a rat model of post-myocardial infarction heart failureCardiovascular Diabetology, 2011
- Sonic hedgehog is a critical mediator of erythropoietin-induced cardiac protection in miceJournal of Clinical Investigation, 2010
- Effects of Sitagliptin Treatment on Dysmetabolism, Inflammation, and Oxidative Stress in an Animal Model of Type 2 Diabetes (ZDF Rat)Mediators of Inflammation, 2010
- Stromal Cell–Derived Factor-1α Is Cardioprotective After Myocardial InfarctionCirculation, 2008
- Stromal Cell–Derived Factor-1α Confers Protection Against Myocardial Ischemia/Reperfusion InjuryCirculation, 2007
- The chemokine receptor CXCR4 is essential for vascularization of the gastrointestinal tractNature, 1998