Elimination of Antigen-Presenting Cells and Autoreactive T Cells by Fas Contributes to Prevention of Autoimmunity
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (AI044130, AI072627)
- Juvenile Diabetes Research Foundation United States of America (546)
This publication has 43 references indexed in Scilit:
- Perforin Gene Defects in Familial Hemophagocytic LymphohistiocytosisScience, 1999
- Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease.Journal of Clinical Investigation, 1996
- Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomasNature, 1995
- Fatal interactions: fas-induced apoptosis of mature T cellsImmunity, 1994
- The mouse fas-ligand gene is mutated in gld mice and is part of a TNF family gene clusterImmunity, 1994
- T cell receptor antagonist peptides induce positive selectionCell, 1994
- Generation of large numbers of dendritic cells from mouse bone marrow cultures supplemented with granulocyte/macrophage colony-stimulating factor.Journal of Experimental Medicine, 1992
- Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosisNature, 1992
- The influence of the lpr gene on B cell activation: Differential antibody expression in lpr congenic mouse strainsClinical Immunology and Immunopathology, 1984
- Mechanisms of polyclonal B-cell activation in autoimmune B6-lpr/lpr miceCellular Immunology, 1984