Matrix Metalloprotease Activity Is Enhanced in the Compensated but Not in the Decompensated Phase of Pressure Overload Hypertrophy
Authors:
Open Access
- 1 June 2007
- journal article
- Published by Oxford University Press (OUP) in American Journal of Hypertension
- Vol. 20 (6) , 663-669
Abstract
Background:. During the transition of pressure overload hypertrophy (POH) to heart failure (HF) there is intense interstitial cardiac remodeling, charaKeywords
This publication has 21 references indexed in Scilit:
- Excessive activation of matrix metalloproteinases coincides with left ventricular remodeling during transition from hypertrophy to heart failure in hypertensive ratsJACC, 2002
- Effects of chronic adenosine uptake blockade on adrenergic responsiveness and left ventricular chamber function in pressure overload hypertrophy in the ratJournal Of Hypertension, 1998
- Pathologic fibrosis and connective tissue matrix in left ventricular hypertrophy due to chronic arterial hypertension in humansJournal Of Hypertension, 1998
- Matrix metalloproteinase activity expression in infarcted, noninfarcted and dilated cardiomyopathic human heartsMolecular and Cellular Biochemistry, 1996
- Myocardial fibrosis in hypertensive heart disease: an overview of potential regulatory machanismsEuropean Heart Journal, 1995
- Quantitative Zymography: Detection of Picogram Quantities of GelatinasesAnalytical Biochemistry, 1994
- Quantitative assessment of myocardial collagen with picrosirius red staining and circularly polarized lightBasic Research in Cardiology, 1994
- Myocardial matrix metalloproteinase(s): localization and activationMolecular and Cellular Biochemistry, 1993
- Reactive and reparative myocardial fibrosis in arterial hypertension in the ratCardiovascular Research, 1992
- PROTEIN MEASUREMENT WITH THE FOLIN PHENOL REAGENTJournal of Biological Chemistry, 1951