E-cigarette aerosol constituents modulate Leydig cell steroidogenic pathways: Evidence from experimental models
- 13 March 2026
- journal article
- review article
- Published by Elsevier in Molecular and Cellular Endocrinology
- Vol. 617, 112786
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Higher Education, Malaysia (FRGS / 1/2020/SKK05/UM/02/1)
This publication has 117 references indexed in Scilit:
- E-Cigarette Nicotine Delivery Among Young Adults by Nicotine Form, Concentration, and FlavorJAMA Network Open, 2024
- Biomarkers of Electronic Nicotine Delivery Systems (ENDS) useAddiction Neuroscience, 2023
- Effect of exogenous testosterone in the context of energy deficit on risky choice: Behavioural and neural evidence from malesBiological Psychology, 2022
- The role of testosterone, the androgen receptor, and hypothalamic-pituitary–gonadal axis in depression in ageing MenReviews in Endocrine and Metabolic Disorders, 2022
- Electronic cigarette aerosols induce oxidative stress-dependent cell death and NF-κB mediated acute lung inflammation in miceArchives of Toxicology, 2020
- Mitochondria as a possible target for nicotine actionJournal of Bioenergetics and Biomembranes, 2019
- Nicotine induced autophagy of Leydig cells rather than apoptosis is the major reason of the decrease of serum testosteroneThe International Journal of Biochemistry & Cell Biology, 2018
- Role of the steroidogenic acute regulatory protein in health and diseaseEndocrine, 2015
- Cortisol levels decrease after acute tobacco abstinence in regular smokersHuman Psychopharmacology: Clinical and Experimental, 2014
- Mitochondrial ROS-induced ROS release: An update and reviewBiochimica et Biophysica Acta (BBA) - Bioenergetics, 2006