Emerging role of astroglia in pain hypersensitivity
Author:
Open Access
- 1 February 2010
- journal article
- review article
- Published by Elsevier in Japanese Dental Science Review
- Vol. 46 (1) , 86-92
Abstract
No abstract availableKeywords
Funding Information
- NIH (DE11964, NS060735)
This publication has 71 references indexed in Scilit:
- Supraspinal Glial–Neuronal Interactions Contribute to Descending Pain FacilitationJournal of Neuroscience, 2008
- IL-1ra alleviates inflammatory hyperalgesia through preventing phosphorylation of NMDA receptor NR-1 subunit in ratsPain, 2008
- Glial–Cytokine–Neuronal Interactions Underlying the Mechanisms of Persistent PainThe Journal of Neuroscience, 2007
- Catechol- O -methyltransferase gene polymorphisms are associated with multiple pain-evoking stimuliPain, 2006
- Astrocyte Control of Synaptic Transmission and Neurovascular CouplingPhysiological Reviews, 2006
- Role of astrocytic S100β in behavioral hypersensitivity in rodent models of neuropathic painNeuroscience, 2006
- Involvement of spinal cord nuclear factor κB activation in rat models of proinflammatory cytokine‐mediated pain facilitationEuropean Journal of Neuroscience, 2005
- Acute cardiac injury induces glial cell response and activates extracellular signaling-regulated kinase-1 and -2 in the spinal cord of Wistar ratsNeuroscience Letters, 2004
- Activation of fibres in rat sciatic nerve alters phosphorylation state of connexin-43 at astrocytic gap junctions in spinal cord: evidence for junction regulation by neuronal–glial interactionsNeuroscience, 2000
- An In Vivo Model for Studying Function of Brain Tissue Temporarily Devoid of Glial Cell Metabolism: The Use of FluorocitrateJournal of Neurochemistry, 1987