CCAAT‐enhancer binding protein delta functions as a tumor suppressor gene in acute myeloid leukemia
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Funding Information
- University of Virginia
- Leukemia and Lymphoma Society
- Government of South Australia
- Starr Foundation (I4-A442)
- National Cancer Institute
- Cancer Research UK
- Blood Cancer UK
- Alexander and Margaret Stewart Trust
- V Foundation for Cancer Research
- National Institutes of Health
- American Society of Hematology (ASHAMFDP-20121)
- Starr Cancer Consortium (LLS SCOR 7006-13)
- National Institute for Health and Care Research
- Cancer Council South Australia
- National Computational Infrastructure (P30 CA44579)
- Pew Charitable Trusts
This publication has 16 references indexed in Scilit:
- Assessing next-generation sequencing-based computational methods for predicting transcriptional regulators with query gene setsBriefings in Bioinformatics, 2024
- The dual role of C/EBPδ in cancerCritical Reviews in Oncology/Hematology, 2023
- C/EBPδ protects from radiation-induced intestinal injury and sepsis by suppression of inflammatory and nitrosative stressScientific Reports, 2019
- E2F4 regulates transcriptional activation in mouse embryonic stem cells independently of the RB familyNature Communications, 2019
- BART: a transcription factor prediction tool with query gene sets or epigenomic profilesBioinformatics, 2018
- C/EBPα deregulation as a paradigm for leukemogenesisLeukemia, 2017
- Targeting MTHFD2 in acute myeloid leukemiaJournal of Experimental Medicine, 2016
- Comparison of promoter DNA methylation and expression levels of genes encoding CCAAT/enhancer binding proteins in AML patientsLeukemia Research, 2014
- C/EBPδ expression in a BCR-ABL-positive cell line induces growth arrest and myeloid differentiationOncogene, 2005
- c-Myc Is a Critical Target for C/EBPα in GranulopoiesisMolecular and Cellular Biology, 2001