Copper dysfunction in Alzheimer's disease: From meta-analysis of biochemical studies to new insight into genetics
- 1 June 2012
- journal article
- review article
- Published by Elsevier in Journal of Trace Elements in Medicine and Biology
- Vol. 26 (2-3) , 93-96
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Copper Hypothesis in the Missing Hereditability of Sporadic Alzheimer's Disease: ATP7B Gene as Potential Harbor of Rare VariantsJournal of Alzheimer’s Disease, 2012
- Alzheimer's disease & metals: therapeutic opportunitiesBritish Journal of Pharmacology, 2011
- Copper transport to the brain by the blood-brain barrier and blood-CSF barrierBrain Research, 2009
- Therapeutics for Alzheimer's Disease Based on the Metal HypothesisNeurotherapeutics, 2008
- Tyrosine gated electron transfer is key to the toxic mechanism of Alzheimer's disease β‐amyloidThe FASEB Journal, 2004
- Wilson's disease: the importance of measuring serum caeruloplasmin non-immunologicallyAnnals of Clinical Biochemistry: International Journal of Laboratory Medicine, 2003
- Metal complexing agents as therapies for Alzheimer’s diseaseNeurobiology of Aging, 2002
- The Alzheimer's Disease Amyloid Precursor Protein Modulates Copper-Induced Toxicity and Oxidative Stress in Primary Neuronal CulturesJournal of Neuroscience, 1999
- Aqueous Dissolution of Alzheimer's Disease Aβ Amyloid Deposits by Biometal DepletionJournal of Biological Chemistry, 1999
- The Amyloid Precursor Protein of Alzheimer's Disease in the Reduction of Copper(II) to Copper(I)Science, 1996