Dimerization of the Human Receptors for Prostacyclin and Thromboxane Facilitates Thromboxane Receptor-mediated cAMP Generation

This publication has 48 references indexed in Scilit:

• Role of Prostacyclin in the Cardiovascular Response to Thromboxane A ₂
  Science, 2002
• Comparison of Upper Gastrointestinal Toxicity of Rofecoxib and Naproxen in Patients with Rheumatoid Arthritis
  New England Journal of Medicine, 2000
• Prostacyclin synthase gene transfer inhibits neointimal formation in rat balloon-injured arteries without bleeding complications
  Cardiovascular Research, 1999
• Tolerance development to antimitogenic actions of prostacyclin but not of prostaglandin E1 in coronary artery smooth muscle cells
  European Journal of Pharmacology, 1998
• Effect of Serotonin, Thromboxane A₂, and Specific Receptor Antagonists on Vascular Smooth Muscle Cell Proliferation
  Circulation, 1997
• Pharmacodynamic profile of prostacyclin
  The American Journal of Cardiology, 1995
• Mechanisms of platelet activation: Thromboxane A2 as an amplifying signal for other agonists
  The American Journal of Cardiology, 1991
• Cultured human vascular smooth muscle cells with functional thromboxane A2 receptors: measurement of U46619-induced 45calcium efflux.
  Circulation Research, 1987
• Prostacyclin: Homeostatic Regulator or Biological Curiosity?
  Clinical Science, 1981
• Thromboxanes: a new group of biologically active compounds derived from prostaglandin endoperoxides.
  Proceedings of the National Academy of Sciences, 1975