Changes in gene expression and assessment of DNA methylation in primary human hepatocytes and HepG2 cells exposed to the environmental contaminants—Hexabromocyclododecane and 17-β oestradiol

This publication has 36 references indexed in Scilit:

• Global genomic methylation levels in the liver and gonads of the three-spine stickleback (Gasterosteus aculeatus) after exposure to hexabromocyclododecane and 17-β oestradiol
  Environment International, 2008
• Oestrogen receptor α is required for biochanin A-induced apolipoprotein A-1 mRNA expression in HepG2 cells
  British Journal of Nutrition, 2007
• Hepatocellular neoplasms after intrahepatic transplantation of ovarian fragments into ovariectomized rats†
  Hepatology, 2006
• Phenobarbital Induces Progressive Patterns of GC-Rich and Gene-Specific Altered DNA Methylation in the Liver of Tumor-Prone B6C3F1 Mice
  Toxicological Sciences, 2006
• Subacute effects of the brominated flame retardants hexabromocyclododecane and tetrabromobisphenol A on hepatic cytochrome P450 levels in rats
  Toxicology, 2006
• Relevance of DNA methylation in the management of cancer
  The Lancet Oncology, 2003
• Methylation meets genomics
  Nature Genetics, 2001
• Cell proliferation and global methylation status changes in mouse liver after phenobarbital and/or choline-devoid, methionine-deficient diet administration
  Carcinogenesis, 1996
• A genomic sequencing protocol that yields a positive display of 5-methylcytosine residues in individual DNA strands.
  Proceedings of the National Academy of Sciences, 1992
• Hypomethylation distinguishes genes of some human cancers from their normal counterparts
  Nature, 1983