The lipid flippase heterodimer ATP8B1–CDC50A is essential for surface expression of the apical sodium-dependent bile acid transporter (SLC10A2/ASBT) in intestinal Caco-2 cells
- 1 December 2014
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
- Vol. 1842 (12) , 2378-2386
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- P4 ATPases: Flippases in Health and DiseaseInternational Journal of Molecular Sciences, 2013
- ATP9B, a P4-ATPase (a Putative Aminophospholipid Translocase), Localizes to the trans-Golgi Network in a CDC50 Protein-independent MannerJournal of Biological Chemistry, 2011
- CDC50 Proteins Are Critical Components of the Human Class-1 P4-ATPase Transport MachineryJournal of Biological Chemistry, 2010
- Heteromeric Interactions Required for Abundance and Subcellular Localization of Human CDC50 Proteins and Class 1 P4-ATPasesJournal of Biological Chemistry, 2010
- Progressive Familial Intrahepatic Cholestasis Type 1Seminars in Liver Disease, 2010
- ATP8B1 requires an accessory protein for endoplasmic reticulum exit and plasma membrane lipid flippase activityHepatology, 2007
- FIC1 Is Expressed at Apical Membranes of Different Epithelial Cells in the Digestive Tract and Is Induced in the Small Intestine During Postnatal Development of MicePediatric Research, 2004
- Characterization of mutations in ATP8B1 associated with hereditary cholestasisHepatology, 2004
- FIC1, the protein affected in two forms of hereditary cholestasis, is localized in the cholangiocyte and the canalicular membrane of the hepatocyteJournal of Hepatology, 2001
- A gene encoding a P-type ATPase mutated in two forms of hereditary cholestasisNature Genetics, 1998