Persistent effect of TGF-β1 on extracellular matrix gene expression in human dermal fibroblasts
- 1 May 1990
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 169 (1) , 232-238
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Evaluation of Transforming Growth Factor β and Type I Procollagen Gene Expression in Fibrotic Skin Disease by In Situ HybridizationJournal of Investigative Dermatology, 1990
- Latent transforming growth factor-beta from human platelets. A high molecular weight complex containing precursor sequences.Journal of Biological Chemistry, 1988
- Modulation of fibroblast functions by interleukin 1: increased steady-state accumulation of type I procollagen messenger RNAs and stimulation of other functions but not chemotaxis by human recombinant interleukin 1 alpha and betaJournal of Cell Biology, 1988
- A nuclear factor 1 binding site mediates the transcriptional activation of a type I collagen promoter by transforming growth factor-βCell, 1988
- Transforming growth factor β (TGFβ) causes a persistent increase in steady-state amounts of type I and type III collagen and fibronectin mRNAs in normal human dermal fibroblastsBiochemical Journal, 1987
- Some recent advances in the chemistry and biology of transforming growth factor-beta.Journal of Cell Biology, 1987
- Transforming growth factor-beta increases steady state levels of type I procollagen and fibronectin messenger RNAs posttranscriptionally in cultured human dermal fibroblasts.Journal of Clinical Investigation, 1987
- Cellular distribution of type I and type II receptors for transforming growth factor-beta.Journal of Biological Chemistry, 1986
- The biology of platelet-derived growth factorCell, 1986
- Characterization of a membrane receptor for transforming growth factor-beta in normal rat kidney fibroblasts.Journal of Biological Chemistry, 1984