Heme induces inflammatory injury by directly binding to the complex of myeloid differentiation protein 2 and toll-like receptor 4
- 14 September 2022
- journal article
- research article
- Published by Elsevier in Toxicology Letters
- Vol. 370, 15-23
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Blockage of ROS and NF-κB-mediated inflammation by a new chalcone L6H9 protects cardiomyocytes from hyperglycemia-induced injuriesBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2015
- Role of Heme in Cardiovascular Physiology and DiseaseJournal of the American Heart Association, 2015
- Haptoglobin, hemopexin, and related defense pathways—basic science, clinical perspectives, and drug developmentFrontiers in Physiology, 2014
- Heme on innate immunity and inflammationFrontiers in Pharmacology, 2014
- Heme triggers TLR4 signaling leading to endothelial cell activation and vaso-occlusion in murine sickle cell diseaseBlood, 2014
- Macrophages and Systemic Iron HomeostasisJournal of Innate Immunity, 2012
- Inhibition of high glucose‐induced inflammatory response and macrophage infiltration by a novel curcumin derivative prevents renal injury in diabetic ratsBritish Journal of Pharmacology, 2012
- Pathogen recognition by innate receptorsJournal of Infection and Chemotherapy, 2008
- Characterization of Heme as Activator of Toll-like Receptor 4Journal of Biological Chemistry, 2007
- Probing Structure-Function Relations in Heme-Containing Oxygenases and PeroxidasesScience, 1988