A novel CCAAT/enhancer binding protein α germline variant in a case of acute myeloid leukemia
- 3 January 2012
- journal article
- Published by Taylor & Francis in Leukemia & Lymphoma
- Vol. 53 (5) , 1006-1007
Abstract
No abstract availableThis publication has 11 references indexed in Scilit:
- Prognostic impact, concurrent genetic mutations, and gene expression features of AML with CEBPA mutations in a cohort of 1182 cytogenetically normal AML patients: further evidence for CEBPA double mutant AML as a distinctive disease entityBlood, 2011
- Prognostic Significance of CEBPA Mutations in a Large Cohort of Younger Adult Patients With Acute Myeloid Leukemia: Impact of Double CEBPA Mutations and the Interaction With FLT3 and NPM1 MutationsJournal of Clinical Oncology, 2010
- Acute Myeloid Leukemia With Biallelic CEBPA Gene Mutations and Normal Karyotype Represents a Distinct Genetic Entity Associated With a Favorable Clinical OutcomeJournal of Clinical Oncology, 2010
- Double CEBPA mutations, but not single CEBPA mutations, define a subgroup of acute myeloid leukemia with a distinctive gene expression profile that is uniquely associated with a favorable outcomeBlood, 2009
- Somatic CEBPA Mutations Are a Frequent Second Event in Families With Germline CEBPA Mutations and Familial Acute Myeloid LeukemiaJournal of Clinical Oncology, 2008
- Another pedigree with familial acute myeloid leukemia and germline CEBPA mutationLeukemia, 2008
- CEBPA polymorphisms and mutations in patients with acute myeloid leukemia, myelodysplastic syndrome, multiple myeloma and non-Hodgkin's lymphomaBlood Cells, Molecules, and Diseases, 2008
- Recurrent in-frame insertion in C/EBPα TAD2 region is a polymorphism without prognostic value in AMLLeukemia, 2007
- Further evidence that germline CEBPA mutations cause dominant inheritance of acute myeloid leukaemiaLeukemia, 2005
- Mutation ofCEBPAin Familial Acute Myeloid LeukemiaNew England Journal of Medicine, 2004