Ginsenoside compound K increases glucagon-like peptide-1 release and L-cell abundance in db/db mice through TGR5/YAP signaling
Authors:
Open Access
- 9 November 2022
- journal article
- research article
- Published by Elsevier in International Immunopharmacology
- Vol. 113 (Pt A) , 109405
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI-H716 cells via bile acid receptor activationArchives of Pharmacal Research, 2014
- Novel Rho/MRTF/SRF Inhibitors Block Matrix-stiffness and TGF-β–Induced Fibrogenesis in Human Colonic MyofibroblastsInflammatory Bowel Diseases, 2014
- A Mechanical Checkpoint Controls Multicellular Growth through YAP/TAZ Regulation by Actin-Processing FactorsCell, 2013
- The Intestinal Crypt, A Prototype Stem Cell CompartmentCell, 2013
- Ras-related C3 botulinum toxin substrate 1 (RAC1) regulates glucose-stimulated insulin secretion via modulation of F-actinDiabetologia, 2013
- TGR5 potentiates GLP-1 secretion in response to anionic exchange resinsScientific Reports, 2012
- The Hippo signaling pathway restricts the oncogenic potential of an intestinal regeneration programGenes & Development, 2010
- Stem Cells, Self-Renewal, and Differentiation in the Intestinal EpitheliumAnnual Review of Physiology, 2009
- Bile Acids Metabonomic Study on the CCl4- and α-Naphthylisothiocyanate-Induced Animal Models: Quantitative Analysis of 22 Bile Acids by Ultraperformance Liquid Chromatography−Mass SpectrometryChemical Research in Toxicology, 2008
- The incretin system: glucagon-like peptide-1 receptor agonists and dipeptidyl peptidase-4 inhibitors in type 2 diabetesThe Lancet, 2006