Prolactin treatment reduces kainic acid-induced gliosis in the hippocampus of ovariectomized female rats
Authors:
- 1 November 2020
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 1746, 147014
Abstract
No abstract availableKeywords
Funding Information
- Universidad Nacional Autónoma de México (IN204718)
- Secretaría de Ciencia, Humanidades, Tecnología e Innovación (429388)
- Secretaría de Ciencia, Humanidades, Tecnología e Innovación (A1S8948)
This publication has 47 references indexed in Scilit:
- BDNF overexpression in mouse hippocampal astrocytes promotes local neurogenesis and elicits anxiolytic-like activitiesTranslational Psychiatry, 2013
- Inhibition of Reactive Gliosis Attenuates Excitotoxicity-Mediated Death of Retinal Ganglion CellsPLOS ONE, 2011
- Microglial Toll-like Receptor 2 Contributes to Kainic Acid-induced Glial Activation and Hippocampal Neuronal Cell DeathJournal of Biological Chemistry, 2010
- Kainic Acid‐Induced Neurodegenerative Model: Potentials and LimitationsBioMed Research International, 2010
- Microglial Activation and Chronic NeurodegenerationNeurotherapeutics, 2010
- 3D reconstruction of histological sections: Application to mammary gland tissueMicroscopy Research and Technique, 2010
- Kainic Acid-Activated Microglia Mediate Increased Excitability of Rat Hippocampal Neurons in vitro and in vivo: Crucial Role of Interleukin-1betaNeuroimmunomodulation, 2009
- Prolactin Prevents Chronic Stress-Induced Decrease of Adult Hippocampal Neurogenesis and Promotes Neuronal FateJournal of Neuroscience, 2009
- Role of microglial IKKβ in kainic acid-induced hippocampal neuronal cell deathBrain, 2008
- Microglial and Astroglial Reactions to Ischemic and Kainic Acid-Induced Lesions of the Adult Rat HippocampusExperimental Neurology, 1993