Circadian clock disruption in the mouse ovary in response to 2,3,7,8-tetrachlorodibenzo-p-dioxin

This publication has 44 references indexed in Scilit:

• The clock genes period 1 and period 2 mediate diurnal rhythms in dioxin-induced Cyp1A1 expression in the mouse mammary gland and liver
  Toxicology Letters, 2010
• Influence of the estrous cycle on clock gene expression in reproductive tissues: Effects of fluctuating ovarian steroid hormone levels
  Steroids, 2010
• Disruption of CLOCK-BMAL1 Transcriptional Activity Is Responsible for Aryl Hydrocarbon Receptor–Mediated Regulation of Period1 Gene
  Toxicological Sciences, 2010
• The role of the aryl hydrocarbon receptor in the female reproductive system
  Biochemical Pharmacology, 2009
• The aryl hydrocarbon receptor cross-talks with multiple signal transduction pathways
  Biochemical Pharmacology, 2009
• Disruption of period gene expression alters the inductive effects of dioxin on the AhR signaling pathway in the mouse liver
  Toxicology and Applied Pharmacology, 2008
• Behavioral Rhythmicity of Mice Lacking AhR and Attenuation of Light-Induced Phase Shift by 2,3,7,8-Tetrachlorodibenzo-p-Dioxin
  Journal of Biological Rhythms, 2008
• Concentration-dependent TCDD elimination kinetics in humans: toxicokinetic modeling for moderately to highly exposed adults from Seveso, Italy, and Vienna, Austria, and impact on dose estimates for the NIOSH cohort
  Journal of Exposure Science & Environmental Epidemiology, 2004
• Characterization of Three Splice Variants and Genomic Organization of the Mouse BMAL1 Gene
  Biochemical and Biophysical Research Communications, 1999
• The basic-helix–loop–helix-PAS orphan MOP3 forms transcriptionally active complexes with circadian and hypoxia factors
  Proceedings of the National Academy of Sciences, 1998