Single CpG site methylation controls estrogen receptor gene transcription and correlates with hormone therapy resistance
- 1 July 2017
- journal article
- Published by Elsevier in The Journal of Steroid Biochemistry and Molecular Biology
- Vol. 171, 209-217
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Science, Sports and Culture of Japan
This publication has 39 references indexed in Scilit:
- Eicosapentaenoic Acid Demethylates a Single CpG That Mediates Expression of Tumor Suppressor CCAAT/Enhancer-binding Protein δ in U937 Leukemia CellsJournal of Biological Chemistry, 2011
- Functional Activation of the Estrogen Receptor-α and Aromatase by the HDAC Inhibitor Entinostat Sensitizes ER-Negative Tumors to LetrozoleCancer Research, 2011
- Comprehensive methylome map of lineage commitment from haematopoietic progenitorsNature, 2010
- Methylation matters: binding of Ets-1 to the demethylated Foxp3 gene contributes to the stabilization of Foxp3 expression in regulatory T cellsJournal of Molecular Medicine, 2010
- Differential methylation of tissue- and cancer-specific CpG island shores distinguishes human induced pluripotent stem cells, embryonic stem cells and fibroblastsNature Genetics, 2009
- The human colon cancer methylome shows similar hypo- and hypermethylation at conserved tissue-specific CpG island shoresNature Genetics, 2009
- Restoration of Tamoxifen Sensitivity in Estrogen Receptor–Negative Breast Cancer Cells: Tamoxifen-Bound Reactivated ER Recruits Distinctive Corepressor ComplexesCancer Research, 2006
- Effects of chemotherapy and hormonal therapy for early breast cancer on recurrence and 15-year survival: an overview of the randomised trialsThe Lancet, 2005
- Estrogens and their receptors in breast cancer progression: a dual role in cancer proliferation and invasionCritical Reviews in Oncology/Hematology, 2004
- Treatment of Breast CancerNew England Journal of Medicine, 1998