Cancer Cells Employ Nuclear Caspase-8 to Overcome the p53-Dependent G2/M Checkpoint through Cleavage of USP28
- 1 March 2020
- journal article
- research article
- Published by Elsevier in Molecular Cell
- Vol. 77 (5) , 970-984.e7
Abstract
No abstract availableKeywords
Funding Information
- Fonds National de la Recherche Luxembourg (BMBF/BM/7643621)
- Bundesministerium für Forschung, Technologie und Raumfahrt (FKZ 031A423A)
- Canadian Institutes of Health Research
- Prostate Cancer Canada (RS2014-02)
This publication has 49 references indexed in Scilit:
- A Dual Role of Caspase-8 in Triggering and Sensing Proliferation-Associated DNA Damage, a Key Determinant of Liver Cancer DevelopmentCancer Cell, 2017
- A New Mode of Mitotic SurveillanceTrends in Cell Biology, 2017
- 53BP1 Integrates DNA Repair and p53-Dependent Cell Fate Decisions via Distinct MechanismsMolecular Cell, 2016
- Apoptotic Caspases in Promoting Cancer: Implications from Their Roles in Development and Tissue Homeostasis2016
- 53BP1 and the LINC Complex Promote Microtubule-Dependent DSB Mobility and DNA RepairCell, 2015
- Analysis of TP53 Mutation Status in Human Cancer Cell Lines: A ReassessmentHuman Mutation, 2014
- Roles of Chk1 in cell biology and cancer therapyInternational Journal of Cancer, 2013
- RIP Kinases at the Crossroads of Cell Death and SurvivalCell, 2009
- Whole chromosome instability and cancer: a complex relationshipTrends in Genetics, 2008
- Radiation induces p53 -dependent cell apoptosis in bladder cancer cells with wild-type- p53 but not in p53 -mutated bladder cancer cellsUrological Research, 2003