Voltage-dependent opening of HCN channels: Facilitation or inhibition by the phytoestrogen, genistein, is determined by the activation status of the cyclic nucleotide gating ring
Authors:
- 11 June 2009
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Biomembranes
- Vol. 1788 (9) , 1939-1949
Abstract
No abstract availableKeywords
This publication has 56 references indexed in Scilit:
- Novel Mechanism for Suppression of Hyperpolarization-activated Cyclic Nucleotide-gated Pacemaker Channels by Receptor-like Tyrosine Phosphatase-αJournal of Biological Chemistry, 2008
- Cortisone dissociates the Shaker family K+ channels from their β subunitsNature Chemical Biology, 2008
- C-terminal Movement during Gating in Cyclic Nucleotide-modulated ChannelsJournal of Biological Chemistry, 2008
- Cardiac pacemaker function of HCN4 channels in mice is confined to embryonic development and requires cyclic AMPThe EMBO Journal, 2008
- Kinetic Relationship between the Voltage Sensor and the Activation Gate in spHCN ChannelsJournal of General Physiology, 2007
- Gating of HCN Channels by Cyclic Nucleotides: Residue Contacts that Underlie Ligand Binding, Selectivity, and EfficacyStructure, 2007
- Structure and Rearrangements in the Carboxy-Terminal Region of SpIH ChannelsStructure, 2007
- Voltage Sensor Movement and cAMP Binding Allosterically Regulate an Inherently Voltage-independent Closed−Open Transition in HCN ChannelsJournal of General Physiology, 2007
- Regulation of Gating and Rundown of HCN Hyperpolarization-activated Channels by Exogenous and Endogenous PIP2Journal of General Physiology, 2006
- [20] Processing of X-ray diffraction data collected in oscillation mode1997