‘Frequent’ Ventricular Bigeminy – A Reversible Cause of Dilated Cardiomyopathy. How Frequent is ‘Frequent’?
Authors:
- 29 November 2006
- journal article
- case report
- Published by Oxford University Press (OUP) in European Journal of Heart Failure
- Vol. 8 (8) , 869-873
Abstract
An interesting development in the field of heart failure has been the link between frequent premature ventricular contractions and cardiomyopathy. We report a patient whose frequent ventricular bigeminy resulted in left ventricular impairment that resolved after the use of non-contact mapping during radiofrequency ablation. A review of the literature regarding possible mechanisms is discussed. For the practicing clinician, the question of ‘frequent’ should be taken in context of symptoms and LV function. A single 24-h Holter monitor may not truly reflect the ectopic load. We recommend that if there is associated LV dysfunction and a causal link to frequent PVCs then suppression with radiofrequency ablation is a safe and effective treatment strategy.Keywords
This publication has 25 references indexed in Scilit:
- Radiofrequency catheter ablation of premature ventricular complexes from right ventricular outflow tract improves left ventricular dilation and clinical status in patients without structural heart diseaseJACC, 2005
- Premature Ventricular Contraction‐Induced Concealed Mechanical Bradycardia and Dilated Cardiomyopathy:Journal of Cardiovascular Electrophysiology, 2005
- Effects of Resynchronization Therapy on Cardiac Function in Pacemaker Patients “Upgraded” to Biventricular DevicesJournal of Cardiovascular Electrophysiology, 2004
- Suppression of Frequent Premature Ventricular Contractions and Improvement of Left Ventricular Function in Patients With Presumed Idiopathic Dilated CardiomyopathyMayo Clinic Proceedings, 1998
- The Cellular Basis of Pacing-Induced Dilated CardiomyopathyCirculation, 1995
- Abnormalities in intracellular calcium regulation and contractile function in myocardium from dogs with pacing-induced heart failure.Journal of Clinical Investigation, 1992
- Myocardial Na+,K+-ATPase in tachycardia induced cardiomyopathyJournal of Molecular and Cellular Cardiology, 1992
- Changes in myocardial blood flow during development of and recovery from tachycardia-induced cardiomyopathy.Circulation, 1992
- Beta-adrenergic receptor-G protein-adenylate cyclase complex in experimental canine congestive heart failure produced by rapid ventricular pacing.Circulation Research, 1991
- Premature ventricular complexes in the absence of identifiable heart disease.Circulation, 1981