Endothelial AMPK activation induces mitochondrial biogenesis and stress adaptation via eNOS-dependent mTORC1 signaling
- 1 May 2016
- journal article
- research article
- Published by Elsevier in Nitric Oxide
- Vol. 55-56, 45-53
Abstract
No abstract availableKeywords
Funding Information
- NHLBI (R01HL092122, R01HL098407)
This publication has 45 references indexed in Scilit:
- AMP-activated protein kinase—an energy sensor that regulates all aspects of cell functionGenes & Development, 2011
- Regulation of Endothelial Function by Mitochondrial Reactive Oxygen SpeciesAntioxidants & Redox Signaling, 2011
- Chronic activation of AMP‐activated protein kinase prevents 20‐hydroxyeicosatetraenoic acid‐induced endothelial dysfunctionClinical and Experimental Pharmacology and Physiology, 2011
- Activation of AMP-activated Protein Kinase by Temozolomide Contributes to Apoptosis in Glioblastoma Cells via p53 Activation and mTORC1 InhibitionJournal of Biological Chemistry, 2010
- Hydrogen peroxide inhibits mTOR signaling by activation of AMPKα leading to apoptosis of neuronal cellsLaboratory Investigation, 2010
- Direct control of mitochondrial function by mTORProceedings of the National Academy of Sciences, 2009
- Functional Heterogeneity of the Bone Marrow Vascular NicheAnnals of the New York Academy of Sciences, 2009
- Suppression of the JNK Pathway by Induction of a Metabolic Stress Response Prevents Vascular Injury and DysfunctionCirculation, 2008
- AMPK Phosphorylation of Raptor Mediates a Metabolic CheckpointMolecular Cell, 2008
- AMP-activated protein kinase mediates VEGF-stimulated endothelial NO productionBiochemical and Biophysical Research Communications, 2007