The transcription factor Twist1 in the distal nephron but not in macrophages propagates aristolochic acid nephropathy
- 1 January 2020
- journal article
- research article
- Published by Elsevier in Kidney International
- Vol. 97 (1) , 119-129
Abstract
No abstract availableKeywords
Funding Information
- National Heart, Lung, and Blood Institute
- U.S. Department of Veterans Affairs
- National Institute of Diabetes and Digestive and Kidney Diseases
This publication has 42 references indexed in Scilit:
- Akt/PKB-Mediated Phosphorylation of Twist1 Promotes Tumor Metastasis via Mediating Cross-Talk between PI3K/Akt and TGF-β Signaling AxesCancer Discovery, 2012
- Normal and disease-related biological functions of Twist1 and underlying molecular mechanismsCell Research, 2011
- Twist1-Induced Invadopodia Formation Promotes Tumor MetastasisCancer Cell, 2011
- Distinct Macrophage Phenotypes Contribute to Kidney Injury and RepairJournal of the American Society of Nephrology, 2011
- Fate Tracing Reveals the Pericyte and Not Epithelial Origin of Myofibroblasts in Kidney FibrosisThe American Journal of Pathology, 2010
- Requirement for Twist1 in frontonasal and skull vault development in the mouse embryoDevelopmental Biology, 2009
- Twist1-Mediated Adriamycin-Induced Epithelial-Mesenchymal Transition Relates to Multidrug Resistance and Invasive Potential in Breast Cancer CellsClinical Cancer Research, 2009
- Twist mediates suppression of inflammation by type I IFNs and AxlJournal of Experimental Medicine, 2006
- The renin-angiotensin system blockade does not prevent renal interstitial fibrosis induced by aristolochic acidsKidney International, 2004
- Chronic aristolochic acid toxicity in rabbits: A model of Chinese herbs nephropathy?Kidney International, 2001