Antibody diversification caused by disrupted mismatch repair and promiscuous DNA polymerases
- 1 February 2016
- journal article
- review article
- Published by Elsevier in DNA Repair
- Vol. 38, 110-116
Abstract
No abstract availableKeywords
This publication has 119 references indexed in Scilit:
- Mismatch-mediated error prone repair at the immunoglobulin genesBiomedicine & Pharmacotherapy, 2011
- Uracil residues dependent on the deaminase AID in immunoglobulin gene variable and switch regionsNature Immunology, 2010
- PMS2 endonuclease activity has distinct biological functions and is essential for genome maintenanceProceedings of the National Academy of Sciences, 2010
- Controlling somatic hypermutation in immunoglobulin variable and switch regionsImmunologic Research, 2010
- AID and Somatic Hypermutation2010
- Ubiquitylated PCNA plays a role in somatic hypermutation and class-switch recombination and is required for meiotic progressionProceedings of the National Academy of Sciences, 2008
- Reevaluation of the role of DNA polymerase θ in somatic hypermutation of immunoglobulin genesDNA Repair, 2008
- The multifaceted mismatch-repair systemNature Reviews Molecular Cell Biology, 2006
- RNA Editing Enzyme APOBEC1 and Some of Its Homologs Can Act as DNA MutatorsMolecular Cell, 2002
- RAD6-dependent DNA repair is linked to modification of PCNA by ubiquitin and SUMONature, 2002