Disruption of Spinal Noradrenergic Activation Delays Recovery of Acute Incision-Induced Hypersensitivity and Increases Spinal Glial Activation in the Rat

This publication has 66 references indexed in Scilit:

• Endogenous analgesic action of the pontospinal noradrenergic system spatially restricts and temporally delays the progression of neuropathic pain following tibial nerve injury
  Pain, 2013
• Depletion of Endogenous Noradrenaline Does Not Prevent Spinal Cord Plasticity Following Peripheral Nerve Injury
  The Journal of Pain, 2012
• Pathophysiology of postoperative pain
  Pain, 2011
• Lack of analgesic efficacy of spinal ondansetron on thermal and mechanical hypersensitivity following spinal nerve ligation in the rat
  Brain Research, 2010
• Contribution of the Chemokine (C-C Motif) Ligand 2 (CCL2) to Mechanical Hypersensitivity after Surgical Incision in Rats
  Anesthesiology, 2010
• A Novel Method to Quantify Histochemical Changes Throughout the Mediolateral Axis of the Substantia Gelatinosa After Spared Nerve Injury: Characterization with TRPV1 and Substance P
  The Journal of Pain, 2010
• The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition
  Pain, 2010
• Unmasking the tonic-aversive state in neuropathic pain
  Nature Neuroscience, 2009
• Coexpression of α_2A‐adrenergic and δ‐opioid receptors in substance P‐containing terminals in rat dorsal horn
  Journal of Comparative Neurology, 2009
• A comparison of spinal Iba1 and GFAP expression in rodent models of acute and chronic pain
  Brain Research, 2008