MCM10 mediates RECQ4 association with MCM2‐7 helicase complex during DNA replication

This publication has 33 references indexed in Scilit:

• Identification and analysis of new proteins involved in the DNA damage response network of Fanconi anemia and Bloom syndrome
  Methods, 2009
• Dual DNA unwinding activities of the Rothmund–Thomson syndrome protein, RECQ4
  The EMBO Journal, 2009
• Roles of RECQ helicases in recombination based DNA repair, genomic stability and aging
  Biogerontology, 2008
• More complexity to the Bloom’s syndrome complex
  Genes & Development, 2008
• Excess MCM proteins protect human cells from replicative stress by licensing backup origins of replication
  Proceedings of the National Academy of Sciences, 2008
• A RECQ5–RNA polymerase II association identified by targeted proteomic analysis of human chromatin
  Proceedings of the National Academy of Sciences, 2008
• Dormant origins licensed by excess Mcm2–7 are required for human cells to survive replicative stress
  Genes & Development, 2007
• Tipin is required for stalled replication forks to resume DNA replication after removal of aphidicolin in Xenopus egg extracts
  Proceedings of the National Academy of Sciences, 2007
• Cdc7-Dbf4 Phosphorylates MCM Proteins via a Docking Site-Mediated Mechanism to Promote S Phase Progression
  Molecular Cell, 2006
• A CDK‐catalysed regulatory phosphorylation for formation of the DNA replication complex Sld2–Dpb11
  The EMBO Journal, 2006