TDP-43 Overexpression Enhances Exon 7 Inclusion during the Survival of Motor Neuron Pre-mRNA Splicing

This publication has 34 references indexed in Scilit:

• hnRNP A1 functions with specificity in repression of SMN2 exon 7 splicing
  Human Molecular Genetics, 2007
• TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis
  Biochemical and Biophysical Research Communications, 2006
• Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis
  Science, 2006
• TDP43 depletion rescues aberrant CFTR exon 9 skipping
  FEBS Letters, 2006
• Tau Exons 2 and 10, Which Are Misregulated in Neurodegenerative Diseases, Are Partly Regulated by Silencers Which Bind a SRp30c·SRp55 Complex That Either Recruits or Antagonizes htra2β1
  Journal of Biological Chemistry, 2005
• An extended inhibitory context causes skipping of exon 7 of SMN2 in spinal muscular atrophy
  Biochemical and Biophysical Research Communications, 2004
• The Regulation and Regulatory Activities of Alternative Splicing of the SMN Gene
  Critical Reviews™ in Eukaryotic Gene Expression, 2004
• SRp30c-dependent stimulation of survival motor neuron (SMN) exon 7 inclusion is facilitated by a direct interaction with hTra2beta1
  Human Molecular Genetics, 2002
• SMN oligomerization defect correlates with spinal muscular atrophy severity
  Nature Genetics, 1998
• hnRNP PROTEINS AND THE BIOGENESIS OF mRNA
  Annual Review of Biochemistry, 1993