The effect of Fibulin-5 on early brain injury after subarachnoid hemorrhage in mice
Open Access
- 6 May 2025
- journal article
- Published by Elsevier in Neurochemistry International
- Vol. 187, 105989
Abstract
No abstract availableKeywords
All Related Versions
This publication has 46 references indexed in Scilit:
- The role of autophagy and apoptosis in early brain injury after subarachnoid hemorrhage: an updated reviewMolecular Biology Reports, 2022
- Aneurysmal Subarachnoid Hemorrhage: the Last DecadeTranslational Stroke Research, 2020
- Anti-Vascular Endothelial Growth Factor Treatment Suppresses Early Brain Injury After Subarachnoid Hemorrhage in MiceMolecular Neurobiology, 2015
- Overexpression of Fibulin-5 Attenuates Ischemia/Reperfusion Injury After Middle Cerebral Artery Occlusion in RatsMolecular Neurobiology, 2015
- What is Early Brain Injury?Translational Stroke Research, 2014
- Characterization of individual mouse cerebrospinal fluid proteomesProteomics, 2014
- Tenascin-C Causes Neuronal Apoptosis After Subarachnoid Hemorrhage in RatsTranslational Stroke Research, 2014
- Fibulin-5, an integrin-binding matricellular protein: its function in development and diseaseJournal of Cell Communication and Signaling, 2009
- Mechanisms of TGF-β-mediated apoptosisCell and tissue research, 2002
- Differential Regulation of Fibulin, Tenascin-C, and Nidogen Expression during Wound Healing of Normal and Glucocorticoid-Treated MiceExperimental Cell Research, 1996