Extracellular signal-regulated kinase, receptor interacting protein, and reactive oxygen species regulate shikonin-induced autophagy in human hepatocellular carcinoma
- 1 September 2014
- journal article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 738, 142-152
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (81273540)
- Ministry of Education of the People's Republic of China (NCET-13–0436)
This publication has 62 references indexed in Scilit:
- The dynamic nature of autophagy in cancerGenes & Development, 2011
- Autophagy-deficient mice develop multiple liver tumorsGenes & Development, 2011
- Management of Hepatocellular Carcinoma: An Update ΔσHepatology, 2011
- Autophagy in tumorigenesis and energy metabolism: friend by day, foe by nightCurrent Opinion in Genetics & Development, 2011
- Antioxidants can inhibit basal autophagy and enhance neurodegeneration in models of polyglutamine diseaseHuman Molecular Genetics, 2010
- Necroptosis as an alternative form of programmed cell deathCurrent Opinion in Cell Biology, 2010
- Autophagy Suppresses Tumorigenesis through Elimination of p62Cell, 2009
- Autophagy suppresses tumor progression by limiting chromosomal instabilityGenes & Development, 2007
- Autophagy promotes tumor cell survival and restricts necrosis, inflammation, and tumorigenesisCancer Cell, 2006
- ROS stress in cancer cells and therapeutic implicationsDrug Resistance Updates, 2004