Knock-out of CD73 delays the onset of HR-negative breast cancer by reprogramming lipid metabolism and is associated with increased tumor mutational burden
Open Access
- 19 September 2024
- journal article
- research article
- Published by Elsevier in Molecular Metabolism
- Vol. 89, 102035
Abstract
No abstract availableKeywords
Funding Information
- Medical University of Gdańsk (664/73-3306/63/351, 71-01424/K16 0007669)
- National Scientific Lead Centers (2/566516/SPUB/SP/2023)
- Ministerstwo Edukacji i Nauki
- National Science Centre Poland (2017/27/B/NZ5/02192)
This publication has 63 references indexed in Scilit:
- Drugs targeting adenosine signaling pathways: A current viewBiomedicine & Pharmacotherapy, 2023
- Advances in Analyzing the Breast Cancer Lipidome and Its Relevance to Disease Progression and TreatmentJournal of Mammary Gland Biology and Neoplasia, 2021
- Estrogen receptor‐low breast cancer: Biology chaos and treatment paradoxCancer Communications, 2021
- History of ectonucleotidases and their role in purinergic signalingBiochemical Pharmacology, 2021
- Loss of progesterone receptor is associated with distinct tyrosine kinase profiles in breast cancerBreast Cancer Research and Treatment, 2020
- Mismatch repair protein loss in breast cancer: clinicopathological associations in a large British Columbia cohortBreast Cancer Research and Treatment, 2019
- Assessing the link between BACH1/FANCJ and MLH1 in DNA crosslink repairEnvironmental and Molecular Mutagenesis, 2010
- Elovl6: a new player in fatty acid metabolism and insulin sensitivityJournal of Molecular Medicine, 2009
- Gene regulation profile reveals consistent anticancer properties of progesterone in hormone-independent breast cancer cells transfected with progesterone receptorInternational Journal of Cancer, 2005
- Role of α Class Glutathione S-Transferases as Antioxidant Enzymes in Rodent TissuesToxicology and Applied Pharmacology, 2002